Flunisolide intranasal

Name: Flunisolide intranasal

Dosing & Uses

Dosage Forms & Strengths

nasal spray

  • 25mcg/actuation
  • 29mcg/actuation

Seasonal Allergic Rhinitis

2 sprays each nostril BID; may increase to TID/QID

Administration

Prime spray by actuating 5-6 times first before use, if it has not been used for >4 days, or if it has been disassembled for cleaning

Dosage Forms & Strengths

nasal spray

  • 25mcg/actuation
  • 29mcg/actuation

Seasonal Allergic Rhinitis

<6 years: Safety and efficacy not established

6-14 years: 1 spray each nostril TID, or 2 sprays each nostril BID

Administration

Prime spray by actuating 5-6 times first before use, if it has not been used for >4 days, or if it has been disassembled for cleaning

Adverse Effects

>10%

Nasal burning/stinging (13-44%)

Aftertaste (8-17%)

1-10%

Nausea

Epistaxis

Nasal dryness

Pharyngitis

Cough increased

<1%

Hoarseness

Nasal ulceration

Abnormal sense of smell

Sinusitis

Vertical growth suppression

Warnings

Contraindications

Hypersensitivity

Do not use with untreated local infection involving the nasal mucosa

Recent nasal surgery/injury

Cautions

Replacement of systemic corticosteroids with topical administration can be accompanied by signs of adrenal insufficiency: some patients may experience withdrawal symptoms

Intranasal corticosteroids may cause a reduction in growth velocity when administered to pediatric patients Rare reports of nasal septal perforation

Temporary or permanent loss of sense of smell or taste reported

Because of the inhibitory effect of corticosteroids on wound healing, a nasal corticosteroid should be used with caution in patients who have experienced recent nasal septal ulcers, recurrent epistaxis, nasal surgery or trauma, until healing has occurred

Systemic corticoid effects typical of Cushing's syndrome are minimal with recommended doses of topical steroids, this potential increases with excessive doses

Corticosteroids are known to cause immunosuppression resulting in increased susceptibility to infection

Pharmacology

Mechanism of Action

Elicits potent glucocorticoid and weak mineralocorticoid effects; provides direct anti-inflammatory actions to nasal mucosa

Absorption

Bioavailability: 50% (intranasal); 20% (PO) due high first-pass liver metabolism

Metabolism

Converted by the liver to the much less active primary metabolite and to glucuronide and sulfate conjugates

Elimination

Half-life: 1-2 hr

Excretion: 50% feces; 50% urine

Patient Handout

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